Search across public literature, clinical trials, biomarkers, pathways, and research metadata. NeuroPathIQ organizes results into transparent, source-linked evidence trails.
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How it works
Type a mechanism, disease, drug, biomarker, or researcher name. Or ask a natural-language question exactly as you'd phrase it in a lab meeting.
PubMed, ClinicalTrials.gov, NIH Reporter, OpenAlex, and FDA data searched simultaneously, normalized to a single schema, ranked by relevance.
Results organized by disease, biomarker, drug, and trial — with paper and trial counts, evidence strength ratings, and source citations on every item.
Evidence is analyzed for cross-disease patterns and mechanism overlaps. AI-assisted summaries are source-backed and clearly labeled.
What NeuropathIQ does
Search any mechanism, disease, drug, or biomarker. Get related diseases with paper and trial counts, related biomarkers, drugs by phase, and active trials — in one structured page.
Every trial targeting a mechanism across all diseases in one sortable table. Cross-disease comparison that doesn't exist anywhere else at this price point.
Fastest-growing pathways, most active biomarkers, new trial activity by mechanism. Factual trend observations from 275M papers — not predictions, not claims.
Step-by-step pathway maps for ALS, Parkinson's, MS, Alzheimer's, and Epilepsy — showing what's been achieved, what's in progress, and where the evidence gaps are.
Search neuroinflammation, mitochondrial dysfunction, protein aggregation, glymphatic clearance, synaptic dysfunction, or microglial activation and instantly see every disease, biomarker, drug, clinical trial, and publication connected to that mechanism.
Permanent reference pages for ALS, Parkinson's, MS, Alzheimer's, Epilepsy, and Huntington's — plus NfL, Tau, GFAP, TSPO, and YKL-40 — each with evidence, trials, and related conditions. Browse diseases →
Data sourced from — all public domain
NeuropathIQ displays titles, authors, abstracts, and DOIs. Full text is linked to the original source. AI insights are derived analysis — not reproduced publisher content.
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